Wall Street Journal
January 15, 1998
New Smoking Study Likely
To Impact Policy, Litigation
By ROBERT LANGRETH
Staff Reporter of THE WALL STREET
JOURNAL
Surprising new findings on the long-term dangers of past cigarette-smoke
exposure will likely alarm many of the 44 million Americans who quit the
habit in the hope of improving
future health.
A giant new study published in the Journal of the American Medical
Association (JAMA) Wednesday found that even decades after people
quit smoking they have a 25% higher amount of artery damage than
people who never smoked.
The new research is expected to have a
powerful effect on policy debates and
future tobacco litigation because it is also
the first major study to show a direct
biological link between secondhand
smoke and artery damage. This result is
expected to spur efforts to ban smoking
in all public places, and may ignite a
whole new wave of liability lawsuits from
people who were exposed to
secondhand smoke through no choice of
their
own.
'Hard
Biological Evidence'
"This is a big-impact study," said Richard
Daynard, a law professor at
Northeastern University and head of the
Tobacco Products Liability Project.
"Now you have hard biological evidence
that [exposure to tobacco smoke] irreversibly damages arteries. The study
likely spells the end of smoking in shared public places in the United
States." Some states, including California, have already banned most
public smoking, and other states will be hard-pressed not to follow, in light
of the new evidence, he predicted.
The research, certain to arm antismoking activists and public health
officials with new ammunition to further restrict smoking, is especially
troubling news for former smokers and those exposed to secondhand
smoke, because there is little they can do to reverse damage they may
have sustained decades ago. Although it is possible for former smokers to
undergo expensive diagnostic tests to identify artery damage, there is no
therapy short of extremely restrictive diets that can consistently reverse
clogged arteries. Clogging or hardening of the arteries leads to heart
attacks and strokes and is the
single leading cause of death in America.
As a result, researchers who performed the new study at Wake Forest
University emphasized the need to prevent teens and other young people
from smoking at all because even a few years of cigarette smoke exposure
may cause permanent damage to arteries that carry blood away from the
heart.
Impact on Artery Hardening
Although scientists long ago realized that smoking can vastly increase the
risk of heart disease and lung cancer, this is the first large study to directly
measure smoking's long-term impact on hardening of the arteries.
Researchers emphasize that the new study should not be interpreted as an
argument against quitting, as the artery damage from smoking is cumulative
and only gets worse the longer
one smokes.
What can former smokers or those exposed to secondhand smoke in the
past do to reduce their risk of disease? Health experts say that while the
damage cannot be easily reversed, the new findings should alert
ex-smokers and those exposed to the smoke from others to the fact that
they are at higher risk for heart disease and stroke. Consequently, they
should pay extra attention to eating a healthy diet, exercising, and keeping
their cholesterol and blood pressure levels down. Heart specialists also
suggest that those exposed to smoke should inform their doctors so their
higher risk can be monitored.
In the study, the researchers followed 10,914 middle-aged men and
women who were either current smokers, former smokers, non-smokers
regularly exposed to secondhand smoke, or non-smokers not regularly
exposed to secondhand smoke. At the beginning of the study, the
researchers measured the thickness of the walls of a major artery in the
neck where hardening often occurs; they performed another measurement
three years later.
Former Smokers' Arteries
The researchers were not surprised to find that in current smokers arteries
thickened 50% faster over a three-year period than they did in people
who had never smoked. More surprising was that the arteries of former
smokers also hardened some 25% faster over the three years than
non-smokers -- indicating the damaging effects to arteries can linger for
decades. Finally, arteries of non-smokers exposed to environmental
tobacco smoke thickened 20% faster than non-smokers with no
secondhand exposure.
Previous research had found that heart disease among current smokers is
two to four times greater than heart disease among those who never
smoked and that among those who quit, the risk drops to one that is 20%
higher than the risk among those who'd never smoked. The new study
explains that this lingering effect is due to permanent and visible damage to
the arteries.
The study hints that a significant portion of the 50% of all cases of heart
disease whose cause is now classified as "unknown" may actually be
caused by secondhand smoke exposure, said Thomas Pearson, a
preventive cardiologist at the University of Rochester who wrote an
editorial in JAMA commenting on the Wake Forest study. "If the current
numbers hold, environmental tobacco smoke may be the third leading
cause of death in the United States,"
he said in an interview.
The new study may propel health agencies to demand additional
compensation from the tobacco industry to help reimburse them for health
costs to people exposed to environmental tobacco smoke. "This could
certainly increase demands for stronger [monetary] concessions from the
tobacco industry," said Richard Hamburg, legislative director for the
American Heart Association.
A spokesman for the Tobacco Institute, a trade organization, said it
wouldn't comment on the study until it performed an independent analysis
of the study.